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Osteoarthritis statistics — quick reference

Last checked: 2026-08-31

Figures below are not pooled across incompatible definitions. Each row preserves source, year, population and method. Confidence intervals are reproduced from the live-retrieved PubMed abstract; “UI” denotes GBD uncertainty interval.

Global burden

Measure Estimate Population / year Method Source
People living with OA 595 million (95% UI 535–656m) Global, 2020 GBD modeled symptomatic radiographic OA, 204 countries/territories GBD 2021 OA Collaborators 2023, PMID 37675071
Share of global population 7.6% (95% UI 6.8–8.4) Global, 2020 Same PMID 37675071
Growth in total cases since 1990 132.2% (95% UI 130.3–134.1) Global, 1990–2020 Same PMID 37675071
Age-standardized YLD rate 255.0 per 100,000 (95% UI 119.7–557.2) Global, 2020 GBD disability weights × severity prevalence PMID 37675071
YLD-rate increase 9.5% (95% UI 8.6–10.1) Global, 1990–2020 Age-standardized PMID 37675071
Rank among causes of YLD, age ≥70 7th Global, 2020 GBD cause ranking PMID 37675071
High-BMI attributable fraction 20.4% (95% UI -1.7 to 36.6) Global, 2020 Comparative risk assessment PMID 37675071

Projected case growth, 2020–2050

Site Projected increase Population Method Source
Knee 74.9% (95% UI 59.4–89.9) Global Mixed-effects forecast PMID 37675071
Hand 48.6% (35.9–67.1) Global Same PMID 37675071
Hip 78.6% (57.7–105.3) Global Same PMID 37675071
Other OA 95.1% (68.1–135.0) Global Same PMID 37675071

Lifetime risk and national snapshots (added 2026-08-31)

Measure Estimate Population / method Source
Lifetime diagnosed symptomatic knee OA 13.83%; 9.60% nonobese men to 23.87% obese women; median diagnosis age 55 US NHIS 2007–08 + OAPol duration model Losina 2013, PMID 23203864
Incidence, ages 55–64 0.37%/year nonobese men to 1.02%/year obese women Same PMID 23203864
Diagnosed by age 60 9.29% of US population Same PMID 23203864
Radiographic knee OA, age ≥60 37.4%; symptomatic radiographic 12.1%; women 42.1% vs men 31.2% radiographic NHANES III Dillon 2006, PMID 17013996
KL 3–4, age ≥60 12.9% women vs 6.5% men NHANES III PMID 17013996
Knee replacement prevalence 1.6% of US adults ≥60 NHANES III PMID 17013996
Lifetime symptomatic hip OA by age 85 25.3% (95% CI 21.3–29.3) Johnston County, n=3,068 Murphy 2010, PMID 20713163
Postindustrial vs early-industrial skeletal knee OA 16% vs 6%; 2.1-fold (1.5–3.1) after age/BMI adjustment US cadaver series Wallace 2017, PMID 28808025
GBD 2017 age-standardized prevalence 3,754.2 per 100,000 (3,389.4–4,187.6) 195 countries Safiri 2020, PMID 32398285
GBD 2017 incidence 181.2 per 100,000 (162.6–202.4) Same PMID 32398285
GBD 2010 knee / hip prevalence 3.8% (3.6–4.1) / 0.85% (0.74–1.02) GBD 2010 Cross 2014, PMID 24553908

Hip-OA prevalence — estimates must retain definition

Estimate Population Definition / method Source year Source
8.55% (95% CI 4.85–13.18) 326,463 participants, 31 studies worldwide Pooled KL grade ≥2 2023 Fan et al., PMID 36991481
1.20% (0.40–2.38) African studies Regional pooled KL-based estimate 2023 PMID 36991481
4.26% (0.02–14.93) Asian studies Same 2023 PMID 36991481
7.95% (1.98–17.36) North American studies Same 2023 PMID 36991481
12.59% (7.17–19.25) European studies Same 2023 PMID 36991481
9.42% (4.81–15.34) Men Pooled sex subgroup 2023 PMID 36991481
7.94% (3.57–13.81) Women Pooled sex subgroup; no significant sex difference 2023 PMID 36991481

Pain–radiograph discordance

Conditional proportion Range across studies Population / method Source
People with knee pain who had radiographic OA 15–76% Systematic search; heterogeneous definitions Bedson et al. 2008, PMID 18764949
People with radiographic knee OA who had pain 15–81% Same PMID 18764949

These ranges are not prevalence estimates to average: they show the consequence of different samples, radiographic thresholds and pain definitions.

Sensitization and neuropathic-like features

Measure Estimate Population / method Source
Pain sensitization prevalence 20% (95% CI 16–26); I²=89% 53 studies, 7,117 knee-OA participants Previtali et al. 2022, PMID 35356833
Local pressure-pain threshold difference SMD -1.00 (95% CI -1.67 to -0.32) Knee OA vs healthy controls PMID 35356833
Distant pressure-pain threshold difference SMD -0.54 (-0.76 to -0.31) Same PMID 35356833
Pressure-pain threshold difference SMD -0.85 (CI -1.1 to -0.6) Earlier QST meta-analysis, knee OA vs controls Fingleton et al. 2015, PMID 25749012
Possible neuropathic-like pain, PainDETECT 40% (95% CI 32–48) Knee OA; questionnaire meta-analysis Zolio et al. 2021, PMID 33971205
Probable neuropathic-like pain, PainDETECT 20% (15–24) Knee OA PMID 33971205
S-LANSS-positive 32% (26–38) Knee OA PMID 33971205
DN4-positive 41% (24–59) Knee OA PMID 33971205
CSI sensitization prevalence 36% (12–59) Knee OA PMID 33971205
Possible neuropathic-like pain 29% (22–37) Hip OA, PainDETECT PMID 33971205
Probable neuropathic-like pain 9% (6–13) Hip OA, PainDETECT PMID 33971205

Questionnaire-positive “neuropathic-like” pain does not establish a nerve lesion; estimates depend strongly on instrument.

Risk estimates

Exposure Estimate Comparator / population Method Source
BMI 25 kg/m² RR 1.59 (95% CI 1.34–1.81) Reference BMI 22.5 Dose-response meta-analysis, 12 studies Zhou et al. 2014, PMID 24990315
BMI 30 kg/m² RR 3.55 (2.51–5.11) Same Same PMID 24990315
BMI 35 kg/m² RR 7.45 (4.19–13.13) Same Same PMID 24990315
Occupational loading overall OR 1.61 (1.45–1.78); I²=83.6% Exposed vs sedentary work; 526,343 people Meta-analysis McWilliams et al. 2011, PMID 21382500
Occupational loading, cohorts OR 1.38 (1.10–1.74) Cohort designs Subgroup meta-analysis PMID 21382500
Occupational loading, cross-sectional OR 1.57 (1.37–1.81) Cross-sectional designs Same PMID 21382500
Occupational loading, case-control OR 1.80 (1.48–2.19) Case-control designs Same; publication bias detected PMID 21382500
OA after ACL injury OR 6.81 (5.70–8.13) ACL injury vs no injury Umbrella review/meta-analysis Webster et al. 2022, PMID 33852440
OA after ACL reconstruction OR 7.7 (6.05–9.79) Reconstruction history vs no injury Same PMID 33852440
OA prevalence ≈10 years after ACL reconstruction 36% (19.70–53.01) Reconstructed cohorts Same; high heterogeneity PMID 33852440

Exercise effects — knee OA

Outcome / time Mean effect on 0–100 scale Evidence base Source
Pain, immediately after treatment 12-point improvement (95% CI 10–15) 44 land-exercise trials Fransen et al. 2015, PMID 26405113
Physical function, immediately 10 points (8–13) Same review PMID 26405113
Quality of life, immediately 4 points (2–5) 13 studies PMID 26405113
Pain, 2–6 months after formal treatment 6 points (3–9) 12 studies PMID 26405113
Function, 2–6 months after formal treatment 3 points (1–5) 10 studies PMID 26405113
Trials with adequate sequence generation, concealment and incomplete-data handling 19/54 (35%) Risk-of-bias assessment PMID 26405113

Intensive diet and exercise (IDEA)

Outcome at 18 months Diet + exercise Diet Exercise Between-group statistic Source
Weight loss 10.6 kg (11.4%) 8.9 kg (9.5%) 1.8 kg (2.0%) Descriptive Messier et al. 2013, PMID 24065013
Knee compressive force 2,487 N 2,687 N Exercise–diet 200 N (95% CI 55–345) PMID 24065013
WOMAC pain (0–20) 3.6 4.8 4.7 Exercise–combined 1.02 (0.33–1.71) PMID 24065013
WOMAC function (0–68) 14.1 18.4 18.4 Exercise–combined 4.29 (2.07–6.50) PMID 24065013
Physical HRQoL 44.7 41.9 Combined–exercise 2.81 (0.86–4.76) PMID 24065013
Retention 399/454 (88%) All groups 18-month RCT PMID 24065013

Semaglutide in obesity-associated knee OA (STEP 9)

Outcome at week 68 Semaglutide Placebo Contrast Source
Body weight change -13.7% -3.2% P<.001 Bliddal et al. 2024, PMID 39476339
WOMAC pain change -41.7 -27.5 P<.001 PMID 39476339
SF-36 physical-function change +12.0 +6.5 P<.001 PMID 39476339
Permanent discontinuation due to adverse events 6.7% 3.0% GI disorders most common PMID 39476339
Baseline profile mean age 56; BMI 40.3; WOMAC pain 70.9; 81.6% women Trial population Limits transfer beyond obesity-associated knee OA PMID 39476339

Medicines

Comparison / outcome Estimate Evidence base Source
Topical NSAID vs acetaminophen, function SMD -0.29 (95% CrI -0.52 to -0.06) Network of 122 RCTs, 47,113 participants Zeng et al. 2021, PMID 34174454
Topical vs oral NSAID, function SMD 0.03 (-0.16 to 0.22) Same PMID 34174454
Topical NSAID vs oral NSAID, GI adverse effects RR 0.46 (0.34–0.61) RCT network PMID 34174454
Oral NSAID regimens with increased AE-withdrawal risk 18.5% of evaluated regimens Network meta-analysis da Costa et al. 2021, PMID 34642179
Opioid regimens with increased AE-withdrawal risk 83.3% Same PMID 34642179
Opioid regimens with increased any-AE risk 89.5% Same PMID 34642179
Tramadol pain absolute improvement 4% (95% CI 3–5) 8 trials, 3,972 participants Toupin April et al. 2019, PMID 31132298
Tramadol adverse events RR 1.34 (1.24–1.46) 4 studies, 2,039 participants PMID 31132298
Tramadol AE withdrawal RR 2.64 (2.17–3.20) 9 studies, 4,533 participants PMID 31132298
Tramadol serious adverse events RR 1.78 (1.11–2.84) 7 studies, 3,612 participants PMID 31132298
Duloxetine pain mean difference -0.88 (95% CI -1.11 to -0.65) 3 RCTs; 992 participants for endpoint Wang et al. 2015, PMID 26176791
Duloxetine ≥30% pain response RR 1.49 (1.31–1.70) 989 participants PMID 26176791
Duloxetine ≥50% pain response RR 1.69 (1.27–2.25) 989 participants PMID 26176791
Duloxetine adverse events RR 2.15 (1.48–3.11) 1,011 participants PMID 26176791

Injections

Intervention / outcome Estimate Population / method Source
Viscosupplementation pain vs placebo SMD -0.08 (95% CI -0.15 to -0.02) 24 large placebo-controlled trials, 8,997 participants Pereira et al. 2022, PMID 36333100
Same translated to 100-mm VAS -2.0 mm (-3.8 to -0.5) Same PMID 36333100
Viscosupplementation serious adverse events RR 1.49 (1.12–1.98) 15 large trials, 6,462 participants PMID 36333100
RESTORE pain change -2.1 PRP vs -1.8 saline 288 participants, 12 months Bennell et al. 2021, PMID 34812863
RESTORE pain contrast -0.4/10 (95% CI -0.9 to 0.2) MCID 1.8 PMID 34812863
RESTORE cartilage-volume contrast -0.2% (-1.9 to 1.5) Medial tibial MRI volume PMID 34812863
RESTORE null secondary outcomes 29/31 Prespecified secondary outcomes PMID 34812863
Repeated triamcinolone cartilage change -0.21 vs -0.10 mm 140 participants; every 12 weeks, 2 years McAlindon et al. 2017, PMID 28510679
Triamcinolone cartilage contrast -0.11 mm (-0.20 to -0.03) MRI index compartment PMID 28510679
Triamcinolone pain contrast -0.6/20 (-1.6 to 0.3) WOMAC pain PMID 28510679
Hip injection network: active treatment vs saline No intervention significantly superior at 2–4 or 6 months 11 RCTs, 1,353 participants Gazendam et al. 2021, PMID 32829298

Procedures and surgery

Outcome Estimate Population / method Source
Sham meniscectomy trial: Lysholm contrast -1.6 (95% CI -7.2 to 4.0) 146 people with degenerative tear, no OA; 12 months Sihvonen et al. 2013, PMID 24369076
WOMET contrast -2.5 (-9.2 to 4.1) Same PMID 24369076
Exercise-pain contrast -0.1/10 (-0.9 to 0.7) Same PMID 24369076
TKR strategy KOOS4 improvement 32.5 vs 16.0 100 surgery-eligible knee-OA patients; 12 months Skou et al. 2015, PMID 26488691
TKR adjusted mean difference 15.8 (95% CI 10.0–21.5) TKR+nonsurgical vs nonsurgical PMID 26488691
Serious adverse events 24 vs 6 (P=.005) Same PMID 26488691
Nonsurgical-to-TKR crossover 13/50 (26%) By 12 months PMID 26488691
UKA vs TKA hospital stay, RCT group -1.20 days (-1.67 to -0.73) Systematic review/meta-analysis Wilson et al. 2019, PMID 30792179
UKA vs TKA 5-year revision RR, trials 5.95 (1.29–27.59) Same PMID 30792179
UKA vs TKA 5-year revision RR, registries 2.50 (1.77–3.54) Same PMID 30792179
UKA vs TKA 5-year revision RR, cohorts 3.13 (1.89–5.17) Same PMID 30792179

Genetics

Finding Estimate Population / method Source
GWAS meta-analysis sample 826,690 people; 177,517 OA 9 populations, 11 phenotypes Boer et al. 2021, PMID 34450027
Independent risk variants 100; 52 previously unreported Same PMID 34450027
SMO rare missense variant and hip OA OR 2.8; frequency 0.11%; P=7.9×10^-12 Iceland + UK Biobank meta-analysis Styrkarsdottir et al. 2018, PMID 30374069
IL11 missense variant and hip OA OR 1.30; frequency 2.08%; P=2.1×10^-11 Same PMID 30374069
COL11A1 common missense variant OR 1.08; frequency 61%; P=5.2×10^-10 Same PMID 30374069
CHADL1 recessive association OR 5.9; P=1.8×10^-25 Same PMID 30374069

Paracetamol, SPACE, glucosamine

Comparison Estimate Evidence base Source
Paracetamol vs placebo, hip/knee pain WMD −3.7/100 (−5.5 to −1.9); not clinically important High-quality GRADE Machado 2015, PMID 25828856
Cochrane paracetamol, pain 3.23/100 better than placebo (5.43 to 1.02 better) 10 RCTs Leopoldino 2019, PMID 30801133
Cochrane paracetamol, function 2.9/100 better (0.95 to 4.89) Same PMID 30801133
SPACE 12-month BPI interference 3.4 opioid vs 3.3 nonopioid (diff 0.1, −0.5 to 0.7) n=240 VA, 97.5% complete Krebs 2018, PMID 29509867
SPACE 12-month BPI severity 4.0 vs 3.5 (diff 0.5, 0.0 to 1.0) favoring nonopioid Same PMID 29509867
GAIT 20% pain-response vs placebo 60.1% Glucosamine +3.9 pp (p=0.30); CS +5.3 (p=0.17); combo +6.5 (p=0.09); celecoxib +10.0 (p=0.008) n=1,583 Clegg 2006, PMID 16495392
Wandel glucosamine/CS/combo vs placebo, 10 cm VAS −0.4 (−0.7 to −0.1) / −0.3 (−0.7 to 0.0) / −0.5 (−0.9 to 0.0) cm; MCID −0.9 cm 10 trials, 3,803 people Wandel 2010, PMID 20847017

NSAID vascular harm

Outcome Estimate Evidence base Source
Coxib major vascular events RR 1.37 (1.14–1.66) 280 placebo trials, 124,513 people CNT 2013, PMID 23726390
Diclofenac major vascular events RR 1.41 (1.12–1.78) Same PMID 23726390
Naproxen major vascular events RR 0.93 (0.69–1.27) Same PMID 23726390
Extra major vascular events ~3 per 1,000 patient-years on coxib or diclofenac, one fatal Same PMID 23726390
Rofecoxib MI vs placebo Rate ratio 2.12 (1.26–3.56) 31 trials, 116,429 people Trelle 2011, PMID 21224324
Real-world MI, 1–7 days naproxen OR 1.53 (1.07–2.33) 446,763 people, 61,460 MIs Bally 2017, PMID 28487435
Real-world MI, 1–7 days diclofenac OR 1.50 (1.06–2.04) Same PMID 28487435

Landmark procedure and DMOAD numbers (added 2026-08-31)

Outcome Estimate Source
Moseley 1-year Knee-Specific Pain Scale Sham 48.9, lavage 54.8, debridement 51.7 Moseley 2002, PMID 12110735
Kirkley 2-year WOMAC Surgery 874 vs control 897 (diff −23, −208 to 161) Kirkley 2008, PMID 18784099
MeTeOR 6-month WOMAC function improvement 20.9 vs 18.5 (diff 2.4, −1.8 to 6.5); 30% PT crossover Katz 2013, PMID 23506518
Kise 2-year KOOS4 Diff 0.9 (−4.3 to 6.1); 19% exercise-to-surgery crossover Kise 2016, PMID 27440192
TOPKAT 5-year OKS Diff 1.04 (−0.42 to 2.50); n=528 Beard 2020, PMID 32369436
Frydendal 6-month Oxford Hip Score THR +15.9 vs training +4.5 (diff 11.4, 8.9–14.0); 21% crossover Frydendal 2024, PMID 39476341
FORWARD 2-year cartilage thickness, 100 µg q6mo vs placebo +0.05 mm (0.03–0.07); no significant WOMAC difference Hochberg 2019, PMID 31593273
Lorecivivint OA-11 Pain NRS week 12 −2.24 vs −2.49 (p=0.185); n=513 Yazici 2025, PMID 39808286
Tanezumab 2.5 / 5 mg composite joint-safety vs NSAID 38.3 (28.0–52.5) / 71.5 (56.7–90.2) vs 14.8 (8.9–24.6) per 1,000 PY Hochberg 2021, PMID 33538113
Tanezumab pooled SC composite joint-safety 145/4,541 (3.2%); 0% placebo, 3.2% 2.5 mg, 6.2% 5 mg, 1.5% NSAID Carrino 2023, PMID 37652258
Methotrexate vs placebo, 52-week VAS pain Diff 0.3 mm (−6.7 to 7.3); n=215 Zhu 2025, PMID 40455462
Septic arthritis after IA GC 0.08% (0.03–0.12) of 14,118 injections Petersen 2019, PMID 31146626
RPOA after hip CSI 6% (3–9%) in studies able to detect it Sabatini 2023, PMID 37941925
GLA:D pain change −12.4 at 3 months, −13.7 at 12 months (0–100); n=9,825 Skou 2017, PMID 28173795
Christensen weight-loss pain ES 0.20 (0 to 0.39) at 6.1 kg (4.7–7.6 kg) Christensen 2007, PMID 17204567
Cam deformity → incident hip OA OR 2.11 (1.55–2.87) Saberi Hosnijeh 2017, PMID 27696746
Acetabular dysplasia → incident hip OA OR 2.19 (1.50–3.21) PMID 27696746
Framingham KL 0 MRI “any abnormality” 89% (631/710); osteophytes 74%; cartilage 69%; BML 52% Guermazi 2012, PMID 22932918
Felson BML in painful vs painless radiographic OA 77.5% vs 30%; large lesions 35.9% vs 2% Felson 2001, PMID 11281736
da Costa 2017 diclofenac 150 mg pain ES −0.57 (−0.69 to −0.45); etoricoxib 60 mg −0.58 (−0.74 to −0.43) da Costa 2017, PMID 28699595
Rutjes large blinded HA trials Pain ES −0.11 (−0.18 to −0.04); unpublished −0.03 (−0.14 to 0.09) Rutjes 2012, PMID 22868835
Hip precaution early dislocation RCT RR 1.8 (0.6–5.2); NRS RR 0.9 (0.3–2.5); n=8,835 Korfitsen 2023, PMID 37039064
Doxycycline 30-month medial JSN 0.30±0.60 vs 0.45±0.70 mm; no pain reduction Brandt 2005, PMID 15986343
ADAMTS5 knockout First single-gene deletion to abrogate murine OA cartilage loss Glasson 2005, PMID 15800624

Known conflicts and caveats

  • GBD estimates are model outputs with uncertainty intervals; they are not a direct census.
  • Radiographic, symptomatic and coded OA prevalence estimates answer different questions.
  • Sensitization and neuropathic-like prevalence varies by questionnaire or QST method; instruments are not interchangeable diagnoses.
  • Network meta-analysis rankings can exaggerate small differences and inherit heterogeneity across dose, comparator and risk of bias.
  • Injection trials have large contextual effects; saline is not equivalent to no treatment.
  • Surgery comparisons combine RCTs, registries and cohorts with different confounding structures; they are displayed separately rather than averaged.
  • Relative risks can appear large when absolute event rates are low; where abstracts did not provide absolute rates, none were invented.
  • GBD 2010, 2017 and 2021 prevalence estimates use expanding case definitions and must not be averaged.
  • Wallace's 2.1-fold modern increase after age/BMI adjustment conflicts with the heuristic that OA growth is “just demography and obesity.”
  • Murphy's null BMI–lifetime-hip-OA association conflicts with incidence studies linking obesity to hip OA.
  • Naproxen is approximately null for major vascular events in CNT RCT IPD (RR 0.93) but not in Bally's real-world MI IPD (OR 1.53 at 1–7 days) — trial vs observational discrepancy, not a number to average.
  • GAIT's overall glucosamine/CS null and moderate–severe subgroup positive result is the opposing pole to Wandel's industry-independent smaller effects.