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Statistics — Chronic kidney disease

Last curated: 2026-09-02 (deepening pass). Every figure carries its source, year, population and method. Conflicting estimates are shown side by side and never averaged.

Global burden

Measure Estimate Source year / population Method Source
Prevalent CKD cases 697.5 million (95% UI 649.2–752.0m) 2017, global GBD modelling GBD CKD Collaboration 2020, PMID 32061315
Global CKD prevalence 9.1% (95% UI 8.5–9.8) 2017, global GBD modelling PMID 32061315
Deaths attributed directly to CKD 1.2 million (95% UI 1.2–1.3m) 2017, global GBD cause-of-death model PMID 32061315
Change in all-age CKD mortality rate +41.5% (95% UI 35.2–46.5); age-standardised rate change not significant (2.8%, −1.5 to 6.3) 1990–2017, global GBD time trend PMID 32061315
Cardiovascular deaths attributable to impaired kidney function 1.4 million (95% UI 1.2–1.6m) 2017, global Comparative risk assessment PMID 32061315

Definition and measurement

Measure Estimate Population Method Source
Duration required for CKD >3 months Global guideline population Consensus definition + evidence review KDIGO 2024, PMID 38490803
G3a eGFR 45–59 mL/min/1.73 m² Adults GFR category PMID 38490803
G3b eGFR 30–44 mL/min/1.73 m² Adults GFR category PMID 38490803
G4 eGFR 15–29 mL/min/1.73 m² Adults GFR category PMID 38490803
G5 eGFR <15 mL/min/1.73 m² Adults GFR category PMID 38490803
A2 albuminuria uACR 30–300 mg/g Adults Albuminuria category PMID 38490803
A3 albuminuria uACR >300 mg/g Adults Albuminuria category PMID 38490803
Race-free equations within 30% of measured GFR ≥85% Validation set, 4,050 participants Equation validation against measured GFR Inker 2021, PMID 34554658
New creatinine-equation median bias, Black participants −3.6 mL/min/1.73 m² Validation set Median eGFR minus measured GFR PMID 34554658
New creatinine-equation median bias, non-Black participants +3.9 mL/min/1.73 m² Validation set Median eGFR minus measured GFR PMID 34554658

Disease-modifying trials

Trial / measure Treatment Control Effect (95% CI) Population / follow-up Source
DAPA-CKD primary composite 9.2% 14.5% HR 0.61 (0.51–0.72); NNT 19 (15–27) 4,304; median 2.4 y PMID 32970396
DAPA-CKD kidney-specific composite HR 0.56 (0.45–0.68) Same trial PMID 32970396
DAPA-CKD all-cause death 4.7% 6.8% HR 0.69 (0.53–0.88) Same trial PMID 32970396
DAPA-CKD total eGFR-slope difference +0.95 (0.63–1.27) mL/min/1.73 m²/y Prespecified analysis PMID 34619108
EMPA-KIDNEY primary composite 13.1% 16.9% HR 0.72 (0.64–0.82) 6,609; median 2.0 y PMID 36331190
EMPA-KIDNEY all-cause hospitalization HR 0.86 (0.78–0.95) Same trial PMID 36331190
EMPA-KIDNEY acute eGFR dip −2.12 (−2.41 to −1.83) mL/min/1.73 m² First 2 months PMID 38061371
EMPA-KIDNEY chronic slope −1.37/y −2.75/y 50% relative reduction (42–58) Prespecified slope analysis PMID 38061371
CREDENCE primary event rate 43.2/1,000 py 61.2/1,000 py HR 0.70 (0.59–0.82) 4,401; median 2.62 y PMID 30990260
CREDENCE kidney-specific composite HR 0.66 (0.53–0.81) Same trial PMID 30990260
FIDELIO kidney composite 17.8% 21.1% HR 0.82 (0.73–0.93) 5,734; median 2.6 y PMID 33264825
FIDELIO hyperkalaemia discontinuation 2.3% 0.9% +1.4 percentage points Same trial PMID 33264825
FIDELITY kidney composite 5.5% 7.1% HR 0.77 (0.67–0.88) 13,026; median 3.0 y PMID 35023547
FIDELITY cardiovascular composite 12.7% 14.4% HR 0.86 (0.78–0.95) Same pooled analysis PMID 35023547
FLOW primary composite 331 events 410 events HR 0.76 (0.66–0.88) 3,533; median 3.4 y PMID 38785209
FLOW eGFR-slope difference +1.16 mL/min/1.73 m²/y Same trial PMID 38785209
FLOW all-cause death HR 0.80 (0.67–0.95) Same trial PMID 38785209
FIND-CKD total eGFR slope (no diabetes) −3.3/y −4.0/y Difference 0.7 (0.3–1.1) mL/min/1.73 m²/y 1,584; 32 months PMID 42246672
FIND-CKD kidney-or-CV composite HR 0.77 (0.60–0.99) Same trial; hierarchical testing PMID 42246672
FIND-CKD hyperkalaemia 17.0% 13.3% Discontinuation 1.5% vs 0.1% Same trial PMID 42246672
FIND-CKD glomerular-disease subgroup, eGFR slope −3.50/y −4.23/y Difference 0.73 (0.22–1.24) mL/min/1.73 m²/y 903 of 1,584; exploratory PMID 42246414
INFINITY pooled kidney composite 22.3/1,000 py 28.8/1,000 py HR 0.76 (0.68–0.86) 14,574 across FIDELIO, FIGARO, FIND-CKD PMID 42248158
INFINITY pooled kidney failure alone HR 0.85 (0.74–0.99) Same pooled analysis PMID 42248158
INFINITY pooled HF hospitalisation or CV death 19.1/1,000 py 23.9/1,000 py HR 0.80 (0.70–0.91) Same pooled analysis PMID 42248158
INFINITY pooled all-cause death HR 0.88 (0.79–0.99) Same pooled analysis PMID 42248158
CONFIDENCE uACR at day 180, combination vs finerenone 29% greater reduction; ratio 0.71 (0.61–0.82) 779 randomized; phase 2, surrogate endpoint PMID 40470996
CONFIDENCE uACR at day 180, combination vs empagliflozin 32% greater reduction; ratio 0.68 (0.59–0.79) Same trial PMID 40470996
CONFIDENCE hyperkalaemia by arm 15.1% combination 18.8% finerenone / 9.7% empagliflozin No significant difference combination vs finerenone Prespecified secondary analysis PMID 41493296
FINE-ONE uACR at 6 months (type 1 diabetes) −34% −12% Ratio 0.75 (0.65–0.87) 242 randomized; surrogate endpoint PMID 41780000
Semaglutide pooled kidney composite 973 events 1,134 events HR 0.84 (0.77–0.91) 30,787 across SELECT, FLOW, SOUL PMID 42567173

Complications, replacement and symptoms

Measure Estimate Population / method Source
TREAT stroke Darbepoetin vs placebo/rescue; HR 1.92 (1.38–2.68) PMID 19880844
EVOLVE primary composite HR 0.93 (0.85–1.02) 3,883 haemodialysis patients; unadjusted ITT PMID 23121374
SHARP major atherosclerotic events RR 0.83 (0.74–0.94) 9,270 CKD patients; median 4.9 y PMID 21663949
IDEAL all-cause death HR 1.04 (0.83–1.30) Early vs late planned dialysis; 828 adults PMID 20581422
IDEAL late-arm start above target 75.9% Started for symptoms above eGFR 7 PMID 20581422
Conservative-care review mortality HR 0.47 (0.34–0.65), favouring dialysis 25 observational studies PMID 34507554
Forgoing-dialysis median survival range 1–41 months 41 cohorts, 5,102 patients PMID 35285915
Forgoing-dialysis hospital admissions 1–2/person-year Cohort synthesis PMID 35285915
Forgoing-dialysis in-hospital days 6–16/person-year Cohort synthesis PMID 35285915
Difelikefalin ≥3-point itch response 49.1% 27.9% placebo; 378 haemodialysis patients PMID 31702883
Difelikefalin ≥4-point itch response 37.1% 17.9% placebo PMID 31702883
AMBER retained on spironolactone 86% 66% placebo; difference 19.5 points (10.0–29.0) PMID 31533906
VALOR-CKD kidney composite HR 0.99 (0.80–1.20) Veverimer vs placebo PMID 38261535
FSGS partial proteinuria remission 42% 26% irbesartan; week 36 PMID 37921461
FSGS total eGFR-slope difference +0.3 (−1.7 to 2.4) mL/min/1.73 m²/y Sparsentan vs irbesartan PMID 37921461

Burden — updated global estimates (GBD 2023 and pooled cohorts)

Measure Estimate Source year / population Method Source
Prevalent CKD, adults ≥20 y 788 million (95% UI 743–843m) 2023, global GBD modelling GBD 2023 CKD Collaborators 2025, PMID 41213283
Prevalent CKD, adults ≥20 y 378 million (354–407m) 1990, global Same model PMID 41213283
Age-standardised adult prevalence 14.2% (13.4–15.2); relative rise 3.5% (2.7–4.1) since 1990 2023, global GBD modelling PMID 41213283
CKD deaths 1.48 million (1.30–1.65m); 9th leading cause 2023, global GBD cause-of-death model PMID 41213283
CKD DALY rate 769.2 (691.8–857.4) per 100,000, age-standardised; 12th leading cause 2023, global GBD PMID 41213283
CV deaths attributable to impaired kidney function 11.5% (8.4–14.5) of all CV deaths 2023, global Comparative risk assessment PMID 41213283
Highest regional age-standardised prevalence 18.0% (16.9–19.4), north Africa and Middle East 2023 GBD PMID 41213283
Stage 1–3 combined prevalence 13.9% (13.1–15.0) of 14.2% 2023, global GBD PMID 41213283
Pooled prevalence, stages 1–5 13.4% (11.7–15.1) ≤2016; 100 studies, 6,908,440 participants Random-effects meta-analysis Hill 2016, PMID 27383068
Pooled prevalence, stages 3–5 10.6% (9.2–12.2) Same Same PMID 27383068
Stage-specific pooled prevalence S1 3.5% (2.8–4.2); S2 3.9% (2.7–5.3); S3 7.6% (6.4–8.9); S4 0.4% (0.3–0.5); S5 0.1% (0.1–0.1) Same Same PMID 27383068
Lifetime risk of dialysis-requiring kidney failure Men 3.14% (3.10–3.18); women 1.42% (1.39–1.44) — ~1 in 32 and 1 in 71 2017, Japan; competing-mortality adjusted National registry life-table Wakasugi 2020, PMID 32040656
Projected CKD population growth at current diagnosis rates +5.8% population, +9.3% costs by 2027 31 countries Microsimulation Wish 2025, PMID 41141496

Measurement — variability, discordance and equation performance

Measure Estimate Population / method Source
Within-person CV, serum creatinine 5.4% (RCV +16%/−14%) 50 stable CKD outpatients, 3 visits <4 weeks Waikar 2018, PMID 30031564
Within-person CV, cystatin C 4.1% (+12%/−11%) Same PMID 30031564
Within-person CV, first-morning uACR 32.5% (+141%/−58%) Same PMID 30031564
Within-person CV, random-spot uACR 29.7% (+124%/−55%) Same PMID 30031564
Within-person CV, random-spot urine albumin concentration 50.6% (+276%/−73%) Same PMID 30031564
Prevalence of eGFRcys ≥30% below eGFRcr, outpatients 11% (cohort range 3–50%) 821,327 outpatients, 23 cohorts Estrella 2025, PMID 41202182
Same, inpatients 35% 39,639 inpatients, 2 cohorts PMID 41202182
Large negative eGFR difference — all-cause mortality 28.4 vs 16.8 per 1,000 py; HR 1.69 (1.57–1.82) Mean 11 y follow-up PMID 41202182
Same — CV mortality / HF / ASCVD / KFRT HR 1.61 (1.48–1.76) / 1.54 (1.40–1.68) / 1.35 (1.27–1.44) / 1.29 (1.13–1.47) Same PMID 41202182
Variance in eGFR difference explained by putative determinants 36% 1,290 CRIC participants with iothalamate GFR McCoy 2025, PMID 40373998
P30 when eGFRcys ≥20% below eGFRcr eGFRcr 50%, eGFRcys 73%, eGFRcr-cys 84% 9,404 paired samples vs iohexol clearance Fu 2023, PMID 36995139
Median bias in the same stratum +15.0 / −8.5 / +0.8 mL/min/1.73 m² Same PMID 36995139
P30 in US mGFR cohorts CKD-EPI 2021 79.2% (78.5–79.9); EKFC race-free 80.1% (79.4–80.7); EKFC population-specific 81.1% (80.5–81.8) 12,854 participants, 9 studies Delanaye 2024, PMID 38101514
Median bias, same cohorts CKD-EPI 2021 +1.22 (0.99–1.47); EKFCPS +0.14 (−0.07 to 0.35) mL/min/1.73 m² Same PMID 38101514
EKFC creatinine equation, adult bias −0.9 mL/min/1.73 m²; 3.1% of estimates in error >30% 11,251 development + 8,378 external validation Pottel 2021, PMID 33166224
Dipstick ≥1+ for uACR ≥30 mg/g Sensitivity 57.8% (54.1–61.4); specificity 95.4% 10,944 Australian adults White 2011, PMID 21411199
Dipstick ≥1+ for uACR ≥300 mg/g Sensitivity 98.9%; specificity 92.6% Same PMID 21411199
Dipstick trace-or-greater for uACR ≥30 / stage A2 Sensitivity 62% / 36%; specificity 88% / 88% 919,383 adults, 33 cohorts Sumida 2020, PMID 32658569
PCR conversion for screening / A2 / A3 Sensitivity 91% / 75% / 87%; specificity 87% / 89% / 98% Same PMID 32658569

Age-adaptation, screening economics and capacity

Measure Estimate Population / method Source
Incidence under fixed vs age-adapted CKD criteria 537 vs 343 new cases per 100,000 person-years Alberta population, 2009–2017 Liu 2021, PMID 34459844
5-year kidney failure / death, fixed vs age-adapted cohorts 1.7% vs 3.0% / 21.9% vs 25.4% Same PMID 34459844
Fixed-criteria-only cases aged ≥65 with eGFR 45–59 and A1 54,342 of 72,703 (75%); 5-year kidney-failure risk ≤0.12% Same PMID 34459844
Ratio of death risk to kidney-failure risk in that group 69× (65–69 y), 122× (70–74), 279× (75–79), 935× (≥80) Same PMID 34459844
ICER, one-time albuminuria screening at age 55 $86,300/QALY; kidney failure −0.29 percentage points; life expectancy 17.29→17.45 y US Markov model, NHANES-calibrated Cusick 2023, PMID 37216661
ICER, 5-yearly screening ages 55–75 with SGLT2i $128,400/QALY; KRT 2.4%→1.9%; +0.13 life-years Same model family Cusick 2024, PMID 39514193
ICER if SGLT2i efficacy 30% lower $145,400–$182,600/QALY for decennial screening Sensitivity analysis PMID 37216661
Dapagliflozin ICER (UK / Germany / Spain) $8,280 / $17,623 / $11,687 per QALY DAPA-CKD lifetime Markov model McEwan 2022, PMID 36323444
Median nephrologists per million population 11.75 (IQR 1.78–24.76) globally; Africa 1.12; South Asia 1.81; Oceania/SE Asia 3.18 ISN-GKHA, 167 countries Okpechi 2024, PMID 39235198
Countries reporting workforce shortages Transplant surgeons 65%; nephrologists 64%; access coordinators 59%; dialysis nurses 58%; interventional radiologists 54% Same PMID 39235198

Surrogate-endpoint performance

Measure Estimate Population / method Source
eGFR slope 0.75 mL/min/1.73 m²/y slower over 2 y → ESKD HR 0.70 (0.68–0.72) at eGFR ≥60; 0.71 (0.68–0.74) at <60 3,758,551 + 122,664 participants, 14 cohorts Grams 2019, PMID 31292199
Absolute 5-year ESKD reduction from that slope change 1.6% (high-risk, 8.3% baseline) vs 0.13% (low-risk, 0.58% baseline) Same PMID 31292199
30% uACR reduction → kidney failure or doubled creatinine 19% lower hazard (95% BCI 5–30%); median R² 0.66 (BCI 0.06–0.98) 48 RCTs, 85,681 participants Heerspink 2026, PMID 41198855
Finerenone: proportion of kidney effect mediated by 4-month uACR change 84% continuous; 64% at the ≥30% threshold 12,512 patients, FIDELIO + FIGARO Agarwal 2023, PMID 38048573
Finerenone: proportion of CV effect so mediated 37% continuous; 26% at threshold Same PMID 38048573

Mechanism and progression

Measure Estimate Population / method Source
Nephron number per kidney, healthy donors 860,000 ± 370,000 1,388 living donors; CT cortical volume × biopsy glomerular density Denic 2017, PMID 28614683
Single-nephron GFR 80 ± 40 nL/min; flat across age <70, sex, height ≤190 cm Same PMID 28614683
Human kidney atlas resolution 51 cell types; 28 injury-associated cell states; >400,000 nuclei/cells; 45 reference + 48 diseased kidneys Multi-omic single-cell and spatial Lake 2023, PMID 37468583
AKI → incident CKD HR 8.8 (3.1–25.5); 25.8 per 100 py 13 cohort studies Coca 2012, PMID 22113526
AKI → new or progressive CKD (consensus definitions) HR 2.67 (1.99–3.58); 17.76 vs 7.59 per 100 py 82 studies, 2,017,437 participants See 2019, PMID 30473140
AKI → ESKD / death HR 4.81 (3.04–7.62) / 1.80 (1.61–2.02) Same PMID 30473140
Allopurinol effect on eGFR change at 104 weeks −0.10 mL/min/1.73 m²/y (−1.18 to 0.97); p=0.85 CKD-FIX, 363 analysed Badve 2020, PMID 32579811
Allopurinol effect on iohexol-measured GFR after washout 0.001 mL/min/1.73 m² (−1.9 to 1.9); p=0.99 PERL, 530 randomized, type 1 diabetes Doria 2020, PMID 32579810
Canakinumab MACE in CKD subgroup HR 0.82 (0.68–1.00); 0.68 (0.53–0.86) if on-treatment hsCRP <2 mg/L 1,875 of 10,061 CANTOS participants Ridker 2018, PMID 29793629
Ambient heat and eGFR −0.6% (−0.9 to −0.3) per 30 days of heat index >30 °C in a 120-day window 4,017 DAPA-CKD participants, 21 countries Zhang 2024, PMID 38580424
Adiposity and GFR decline (BMI 30/35/40 vs 25) HR 1.18 (1.09–1.27) / 1.69 (1.51–1.89) / 2.02 (1.80–2.27); attenuated to 1.03/1.28/1.46 after comorbidity adjustment 5,459,014 adults, 39 cohorts, 40 countries Chang 2019, PMID 30630856

Aetiology

Measure Estimate Population / method Source
APOL1 and FSGS / hypertension-attributed ESKD OR 10.5 (6.0–18.4) / 7.3 (5.6–9.5) African American case-control Genovese 2010, PMID 20647424
Microalbuminuria without diabetes by APOL1 status 2.3% European American; 6.0% AA 0–1 allele; 16.5% AA 2 alleles Dallas Heart Study, 1,825 + 1,042 Friedman 2011, PMID 21997396
eGFR <60 without diabetes by APOL1 status 1.5% / 1.7% / 6.7% Same PMID 21997396
Sickle cell trait and incident ESRD HR 2.03 (1.44–2.84); 8.5 vs 4.0 per 1,000 py REGARDS, 9,909 Black participants Naik 2017, PMID 28280138
APOL1 high-risk genotype in same cohort HR 1.77 (1.31–2.38); 6.6 per 1,000 py Same PMID 28280138
Exome sequencing diagnostic yield in adult CKD 9.3% (307/3,315) across 66 disorders; 23.9% congenital/cystic; 17.1% unknown-origin nephropathy 2 cohorts Groopman 2019, PMID 30586318
Non-diabetic kidney disease at biopsy in diabetes 58.8% (35.9% without concurrent DN; 22.9% as second diagnosis); 2.56-fold lower ESKD risk 49,075 biopsied patients, 2001–2024 Caza 2026, PMID 42034202
Obesity-related glomerulopathy in a biopsy series 0.89% (90/10,093), rising 0.62%→1.0% over 5 years Nanjing, 2002–2006 Chen 2008, PMID 18423814
PPI use and incident CKD HR 1.50 (1.14–1.96) adjusted 10,482 ARIC; replicated in 248,751 Lazarus 2016, PMID 26752337
Lithium and CKD 0.012 cases per exposed patient-year; eGFR decline 1.8 mL/min/y in those reaching G3; no ESKD 1,012 patients, 2000–2015 Van Alphen 2021, PMID 33392830
Primary tubulointerstitial disease among biopsied CKDu referrals 43/87 (49%); 30% with moderate-severe active disease Sri Lanka, 600 new referrals over 1 year Anand 2019, PMID 30659059
Harvest-season eGFR fall in highest-biomarker sugarcane workers 4.6 (1.0–8.2) mL/min/1.73 m² 284 Nicaraguan workers Laws 2016, PMID 26454687

Therapy — pooled class effects

Measure Estimate Population / method Source
SGLT2i and kidney disease progression RR 0.63 (0.58–0.69); similar with and without diabetes 13 trials, 90,409 participants Nuffield Renal Studies Group 2022, PMID 36351458
SGLT2i kidney progression, by diabetes status 33 vs 48 per 1,000 py, HR 0.65 (0.60–0.70) with diabetes; 32 vs 46, HR 0.74 (0.63–0.85) without 8 trials, 58,816 participants Staplin 2026, PMID 41202026
SGLT2i and acute kidney injury 14 vs 18 per 1,000 py, HR 0.77 (0.69–0.87) with diabetes; 13 vs 18, HR 0.72 (0.56–0.92) without Same PMID 41202026
SGLT2i vs other glucose-lowering classes for AKI OR 0.76 (0.66–0.88) vs placebo; 0.79 (0.65–0.97) vs GLP-1 RA; 0.68 (0.54–0.86) vs DPP-4i 20 trials, 156,690 participants Zhao 2020, PMID 33376101
Empagliflozin and serum uric acid −25.6 µmol/L (−30.3 to −21.0); gout events HR 0.87 (0.74–1.02) 6,609 EMPA-KIDNEY participants Mayne 2025, PMID 39277784
Semaglutide kidney composite without diabetes 1.8% vs 2.2%; HR 0.78 (0.63–0.96) 17,604 SELECT participants Colhoun 2024, PMID 38796653
GLP-1 RA MACE by background SGLT2i use HR 0.77 (0.54–1.09) with vs 0.79 (0.71–0.87) without; p-heterogeneity 0.78 3 trials, 17,072 participants (1,743 exposed) Neuen 2024, PMID 39210781
Intensive glucose control and kidney events HR 0.80 (0.72–0.88) for −0.90% HbA1c ACCORD, ADVANCE, UKPDS, VADT; 27,049 Zoungas 2017, PMID 28365411
BP lowering, 5 mmHg systolic, CV events HR 0.91 (0.87–0.94) with CKD; 0.90 (0.88–0.93) without; p-interaction >0.99 46 trials, 285,124 participants Zeng 2026, PMID 42035778
Same, CKD with vs without diabetes HR 0.96 (0.90–1.02) vs 0.88 (0.84–0.93); p-interaction 0.044 Same PMID 42035778
Chlorthalidone, 24-h ambulatory systolic BP at 12 weeks −10.5 mmHg (−14.6 to −6.4) vs placebo CLICK, 160 patients, mean eGFR 23.2 Agarwal 2021, PMID 34739197
Benazepril in advanced CKD (creatinine 3.1–5.0 mg/dL) Composite 41% vs 60%; 43% RRR (p=0.005) 224 patients, mean 3.4 y Hou 2006, PMID 16407508
RAS-inhibitor withdrawal at eGFR <30 eGFR difference −0.7 (−2.5 to 1.0); ESKD/KRT HR 1.28 (0.99–1.65) STOP-ACEi, 411 patients Bhandari 2022, PMID 36326117
Finerenone hyperkalaemia, mild / moderate 21.4% vs 9.2% / 4.5% vs 1.4% over median 2.6 y FIDELIO-DKD Agarwal 2022, PMID 34732509

Complications and replacement — additional figures

Measure Estimate Population / method Source
PIVOTAL primary composite, high- vs low-dose IV iron 29.3% vs 32.3%; HR 0.85 (0.73–1.00) 2,141 haemodialysis patients, median 2.1 y Macdougall 2019, PMID 30365356
PIVOTAL monthly ESA dose difference −7,539 IU (−9,485 to −5,582) Same PMID 30365356
PIVOTAL infection rates, high vs low dose 46.5% vs 45.5% (63.3 vs 69.4 per 100 py); no dose effect Prespecified secondary analysis Macdougall 2020, PMID 32253271
FIND-CKD escalation endpoint 23.5% high-ferritin IV FCM vs 31.8% oral iron; HR 0.65 (0.44–0.95) 626 non-dialysis CKD patients Macdougall 2014, PMID 24891437
Daprodustat vs ESA, first HF hospitalisation HR 1.22 (0.95–1.56) non-dialysis; 1.10 (0.84–1.45) dialysis ASCEND-ND + ASCEND-D, 6,836 Cunningham 2024, PMID 38383961
HIF-PHI vs ESA, MACE RR 0.99 (0.92–1.08) dialysis; 1.08 (0.95–1.22) non-dialysis 25 trials, 26,478 participants Ha 2024, PMID 39186635
LANDMARK cardiovascular events, lanthanum vs calcium carbonate 4.80 vs 4.30 per 100 py; HR 1.11 (0.88–1.41) 2,309 haemodialysis patients, median 3.16 y Ogata 2021, PMID 34003226
PRIMO left ventricular mass index change +0.34 (−0.14 to 0.83) paricalcitol vs −0.07 (−0.55 to 0.42) placebo g/m^2.7 227 patients, 48 weeks Thadhani 2012, PMID 22337679
AURORA primary composite, rosuvastatin on dialysis 9.2 vs 9.5 per 100 py; HR 0.96 (0.84–1.11) despite 43% LDL fall 2,776 haemodialysis patients, median 3.8 y Fellström 2009, PMID 19332456
ISCHEMIA-CKD death or MI 36.4% vs 36.7% at 3 y; adjusted HR 1.01 (0.79–1.29) 777 advanced-CKD patients Bangalore 2020, PMID 32227756
ISCHEMIA-CKD stroke / death-or-dialysis with invasive strategy HR 3.76 (1.52–9.32) / 1.48 (1.04–2.11) Same PMID 32227756
Incident vs progressive coronary calcification Incident CAC: ASCVD HR 2.42 (1.23–4.79), death 1.82 (1.03–3.22); progression ≥50 units/y: ASCVD 1.42 (0.85–2.35), death 1.73 (1.31–2.28) 1,310 CRIC participants Tian 2025, PMID 39154888
Haemodiafiltration vs haemodialysis, all-cause death 23.3% vs 27.0%; HR 0.84 (0.74–0.95); graded convection-volume response IPD meta-analysis, 5 RCTs, 4,153 patients Vernooij 2024, PMID 39489903
Cooler vs standard dialysate, CV composite 21.4% vs 22.4%; adjusted HR 1.00 (96% CI 0.89–1.11) MyTEMP, 84 centres, 15,413 patients MyTEMP writing committee 2022, PMID 36343653
Transplantation vs remaining waitlisted, all-cause death HR 0.45 (0.39–0.54); 11 of 48 studies identified strata without benefit 48 observational studies, 1,245,850 patients Chaudhry 2022, PMID 35232772
PD vs HD from day 0 / from day 90 HR 0.92 (0.86–1.00) / 1.05 (0.96–1.16) 6,337 matched pairs, US 2003 incident cohort Weinhandl 2010, PMID 20133483
RAASi discontinuation after hyperkalaemia — death HR 1.32 (1.22–1.41) Manitoba; 1.47 (1.41–1.52) Ontario 7,200 + 71,290 CKD patients Leon 2022, PMID 35085685
Same — dialysis initiation HR 1.65 (1.41–1.85) / 1.11 (1.08–1.16) Same PMID 35085685
SZC in haemodialysis — arrhythmic composite 8.8% vs 8.9%; HR 0.98 (0.76–1.26) despite normokalaemia OR 3.36 (2.64–4.26) DIALIZE-Outcomes, 2,690, stopped early Fishbane 2025, PMID 40618849
Conservative care vs dialysis pathway — hospital-free days 352.7 vs 282.7 per year; adjusted IRR 1.15 (1.09–1.21) 366 patients ≥70 y, Netherlands Verberne 2018, PMID 30115028
Same — annual treatment cost Adjusted cost ratio 0.43 (0.28–0.67) Same PMID 30115028
Dialysis withdrawal before death 262 of 536 deaths (49%); median 7 days (IQR 4–11) to death; 34% received palliative care 1,226 incident haemodialysis patients Chen 2018, PMID 30026285

Symptoms, function and glomerular disease

Measure Estimate Population / method Source
Fatigue prevalence, CKD not on KRT 70% (60–79) 14 studies, 4,139 participants Fletcher 2022, PMID 35385471
Instruments used in the CKD symptom literature 67 measures across 449 studies covering 68 symptoms, 199,147 participants, 62 countries Systematic review PMID 35385471
Intradialytic exercise, 60-s sit-to-stand at 12 months +3.85 repetitions (2.22–5.48); 6-min walk +37.5 m (14.7–60.4); hospital days 2 vs 5/year 917 analysed of 1,211 randomized Anding-Rost 2023, PMID 38320198
Pre-dialysis supervised exercise VO2peak 17.9 ± 5.5 vs 15.9 ± 7.0 mL/kg/min at 6 months, not sustained at 12; 6MWT +98 feet 99 adults, eGFR 15–<45 Weiner 2023, PMID 35944747
Sertraline vs CBT for depression on haemodialysis QIDS-C difference −1.84 (−3.54 to −0.13) at 12 weeks; more adverse events 120 patients, phase 2 of 41-facility trial Mehrotra 2019, PMID 30802897
Treatment acceptance with vs without engagement interview 66% vs 64% (risk difference 2.1, −12.1 to 16.4) 184 patients, phase 1 PMID 30802897
Frailty prevalence in CKD Frail 41.8% (range 2.8–81.5%); pre-frail 43.9% (19.1–62.7%) 18 cohorts, 22,788 participants Mei 2021, PMID 33218914
Frailty and mortality Pre-frailty HR 1.68 (1.46–1.94); frailty 1.48 (1.21–1.81) Same PMID 33218914
Iptacopan, annualized total eGFR slope −3.10 vs −6.12 mL/min/1.73 m²/y; difference 3.02 (2.02–4.01) APPLAUSE-IgAN, 477 patients, 24 months Barratt 2026, PMID 41910396
Iptacopan, composite kidney failure endpoint 21.4% vs 33.5%; HR 0.57 (0.40–0.81) Same PMID 41910396
Iptacopan, 24-h uPCR at 9 months −38.3% (26.0–48.6) vs placebo Prespecified interim, first 250 randomized Perkovic 2025, PMID 39453772
Ravulizumab, urine protein at 26 weeks −41.9% (−50.2 to −32.0) vs −16.8% (−31.8 to +1.6); 30.1% treatment effect (p=0.005) Phase 2, 66 patients Lafayette 2025, PMID 39455063
Atrasentan, eGFR change at week 136 −7.5 (−9.2 to −5.8) vs −9.9 (−11.7 to −8.1); difference 2.4 (−0.1 to 4.8), p=0.057; total slope difference 1.4/y (0.5–2.3) ALIGN, 404 randomized Heerspink 2026, PMID 42242268
Tolvaptan, total kidney volume growth 2.8%/y (2.5–3.1) vs 5.5%/y (5.1–6.0) TEMPO 3:4, 1,445 patients Torres 2012, PMID 23121377
Tolvaptan, eGFR change over 1 year in later-stage ADPKD −2.34 (−2.81 to −1.87) vs −3.61 (−4.08 to −3.14); difference 1.27 (0.86–1.68) REPRISE, 1,370 patients Torres 2017, PMID 29105594
Rituximab vs cyclosporine remission, membranous nephropathy 60% vs 52% at 12 months; 60% vs 20% at 24 months (difference 40 points, 25–55) MENTOR, 130 patients Fervenza 2019, PMID 31269364
Voclosporin complete renal response at 52 weeks 41% (73/179) vs placebo AURORA 1, 357 patients, 27 countries Rovin 2021, PMID 33971155
Avacopan vs prednisone taper, relapse after remission 9.4% vs 20.9%; HR 0.43 (0.22–0.85) ADVOCATE kidney subgroup, 268 patients Geetha 2025, PMID 40814647

Safety and negative-result figures

Measure Estimate Population / method Source
Clinically significant contrast-associated AKI 1.2% (53/4,418); CA-AKI → 90-day death/dialysis/persistent impairment OR 3.93 (2.82–5.49) PRESERVE cohort Weisbord 2020, PMID 32192658
Bicarbonate vs saline / acetylcysteine vs placebo prophylaxis OR 0.93 (0.72–1.22) / 1.02 (0.78–1.33) PRESERVE, 4,993 analysed Weisbord 2018, PMID 29130810
Nephrogenic systemic fibrosis, group II gadolinium in G4–G5 0 of 4,931 (0%; upper 95% bound 0.07%) 16 studies Woolen 2020, PMID 31816007
Chronic NSAID use and CKD Pooled OR 1.24 (1.11–1.39); HR 1.50 (1.31–1.70); HR 1.67 (1.38–2.02) with pre-existing CKD; I² >90% 40 studies, 1,757,118 participants Soliman 2025, PMID 39412516
Metformin at creatinine >530 µmol/L Mortality 53% vs 41%; adjusted HR 1.35 (1.20–1.51), dose-dependent 813 matched to 2,439, Taiwan Hung 2015, PMID 26094107
Increased water intake, eGFR at 12 months Urine volume +0.6 L/day (0.5–0.7); eGFR difference −0.3 (−1.8 to 1.2) CKD WIT, 631 patients Clark 2018, PMID 29801012
Ultra-processed food and incident CKD Highest vs lowest quartile HR 1.24 (1.15–1.35); 16.5 vs 14.7 per 1,000 py 14,679 ARIC participants, median 24 y Du 2022, PMID 35679994
Ultra-processed food and CKD progression Tertile 3 vs 1 HR 1.22 (1.04–1.42); 2.61 (1.32–5.18) at eGFR ≥60; null below 60 (p-interaction 0.003) 1,047 events, CRIC Sullivan 2023, PMID 37028638
Salt restriction (−80 mEq/day) Clinic SBP −4.9 mmHg (3.1–6.8); proteinuria −0.39 g/day (0.22–0.55) 11 RCTs, 738 patients Garofalo 2018, PMID 29882800
MDRD protein restriction, kidney failure HR 0.89 (0.71–1.12) overall; 0.68 (0.51–0.93) first 6 y; 1.27 (0.90–1.80) after (interaction p=0.008) 585 Study A participants, registry follow-up Levey 2006, PMID 17162142
Achieved protein intake and GFR decline 0.2 g/kg/day lower intake ↔ 1.15 mL/min/y slower decline (p=0.011) 255 Study B participants Levey 1996, PMID 8629624
Bicarbonate vs standard care, creatinine doubling / death 6.6% vs 17.0% (p<0.001) / 3.1% vs 6.8% (p=0.004) UBI, 740 patients, open-label, 36 months Di Iorio 2019, PMID 31598912
Veverimer achieved bicarbonate separation ~1 mEq/L (22.0 ± 3.0 vs 20.9 ± 3.3 at month 3) VALOR-CKD, 1,480 patients Tangri 2024, PMID 38261535

Known conflicts and caveats

  • Prevalence based on a single eGFR or uACR does not prove three-month chronicity and will differ from claims-based diagnosed prevalence.
  • GBD values are modelled estimates with uncertainty intervals; they should not be combined with registry counts as though methods were identical (PMID 32061315).
  • Composite outcomes differ across DAPA-CKD, EMPA-KIDNEY, CREDENCE, FIDELIO and FLOW; relative effects are not a league table.
  • Early-stopped trials can overestimate effects; DAPA-CKD, CREDENCE and FLOW stopped early after prespecified review.
  • Dialysis-versus-conservative-care comparisons are observational and strongly selected; pooled hazard ratios are not causal treatment effects (PMID 34507554).
  • eGFR slope can separate sooner than kidney failure but is sensitive to acute haemodynamic effects (PMIDs: 34619108, 38061371).
  • Albuminuria reduction can fail to translate to eGFR benefit, as the FSGS DUPLEX result demonstrates (PMID 37921461). This caveat applies directly to CONFIDENCE and FINE-ONE, whose endpoints are albuminuria (PMIDs: 40470996, 41780000).
  • FIND-CKD's primary endpoint was total eGFR slope, not a clinical event count; its kidney-only composite hazard ratio was 0.78 (0.60–1.01) and crossed 1.0 (PMID 42246672).
  • The FIND-CKD glomerular-disease analysis is a prespecified exploratory subgroup of 903 participants, not a trial in glomerular disease (PMID 42246414).
  • CONFIDENCE hyperkalaemia percentages are denominators of ~259–265 per arm, not the full randomized set (PMID 41493296).
  • The 2017 and 2023 GBD CKD estimates use different age denominators (all ages versus adults ≥20 y) and are not a time series; only the within-model 1990-versus-2023 comparison in PMID 41213283 is internally consistent.
  • Pooled KFRE discrimination of 0.90 comes from multinational data with wide case-mix; single-country validations give C-statistics near 0.75–0.85 and calibration failures in both directions (PMIDs: 26757465, 38689834, 38184316).
  • Frailty prevalence in CKD ranges 2.8–81.5% because instruments differ; the pooled pre-frailty mortality hazard exceeding the frailty hazard is biologically implausible and indicates residual heterogeneity (PMID 33218914).
  • Screening ICERs are model outputs conditioned on US prices and on SGLT2 inhibitor efficacy imported from one trial; transportability is untested (PMIDs: 37216661, 39514193).
  • Two urate-lowering trials with unambiguous target engagement — one using iohexol-measured GFR — found no effect on kidney function, so the strong urate–progression association is not evidence of a modifiable target (PMIDs: 32579811, 32579810).
  • Proteinuria surrogacy is disease- and mechanism-specific: it translated to eGFR slope and kidney failure in IgA nephropathy (PMID 41910396) and did not in FSGS (PMID 37921461).
  • Modality and transplantation comparisons rest on 2 randomized trials among 84 studies (PMID 38899545) and on 48 observational studies with unresolved heterogeneity (PMID 35232772); hazard ratios are not treatment effects.
  • Finerenone hyperkalaemia rates were obtained under protocol-scheduled potassium measurement with drug withholding and restart, and do not describe unmonitored use (PMID 34732509).
  • Contrast and gadolinium risks are now small in absolute terms; a relative risk of 3.93 attaches to a 1.2% absolute clinically significant event rate (PMID 32192658), and no group II gadolinium NSF case was observed in 4,931 patients with G4–G5 (PMID 31816007).